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Which patient groups were included in sapropterin trials?

See the DrugPatentWatch profile for sapropterin

Main Patient Groups in Sapropterin Trials


Sapropterin (Kuvan), approved for phenylketonuria (PKU), was tested primarily in patients with hyperphenylalaninemia due to tetrahydrobiopterin (BH4)-responsive PKU. Pivotal trials focused on individuals aged 4 years and older with blood phenylalanine (Phe) levels between 10 and 30 mg/dL who showed at least 30% Phe reduction after a BH4 challenge.[1][2]

Key phase 3 trials (PKU-004 and PKU-006) enrolled:
- Children (4-12 years): 89 patients in PKU-004, assessing Phe control over 6 weeks and 10 years long-term.
- Adolescents and adults (13+ years): 245 patients in PKU-006, evaluating 6-week efficacy and long-term safety.[2]

Earlier studies like PKU-001 and PKU-002 included smaller groups of children and adults responsive to BH4 loading tests.[1]

Trials in Infants and Toddlers


A dedicated phase 3 trial (PKU-015) targeted BH4-responsive infants aged 1-3 years with Phe 10-30 mg/dL. It enrolled 19 patients, showing sustained Phe reduction over 4 weeks, with extensions up to 6 years. This group had milder hyperphenylalaninemia at baseline.[2][3]

Patients with Severe or Classical PKU


Most trials excluded severe classical PKU (Phe >30 mg/dL unresponsive to BH4). Responsive patients typically had milder mutations allowing partial BH4 cofactor activity. Subgroup analyses showed better response in those with specific PAH genotypes.[1][4]

Exclusion Criteria Across Trials


Common exclusions: Phe <10 mg/dL or >30 mg/dL, non-BH4 responsive, pregnancy, severe organ issues, or concurrent drugs affecting Phe. Trials required confirmed PKU diagnosis via newborn screening or genotyping.[2]

Long-Term Data on Patient Demographics


Open-label extensions tracked over 1,000 patients (ages 0-50+), with 60% children/adolescents at entry. Demographics skewed female (55-60%) and white (90%+), reflecting PKU prevalence.[2][5]

[1]: FDA Label for Kuvan
[2]: BioMarin Clinical Trial Summaries (PKU Studies)
[3]: Trefz et al., Mol Genet Metab 2011;102:18-23
[4]: Burton et al., Mol Genet Metab 2007;92:63-70
[5]: Long-term registry data, Levy et al., Pediatrics 2013;132:e268-e274



Other Questions About Sapropterin :

Which pku symptoms improve with sapropterin? How do biomarkers distinguish sapropterin responders? Can you name a disease often managed with sapropterin? Is rapid biomarker modification with sapropterin sustained? Which patient groups were primary subjects in sapropterin studies? In what year did sapropterin receive approval? What condition primarily benefits from sapropterin therapy?




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