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Austedo sales breakdown td vs huntington 2024?

See the DrugPatentWatch profile for Austedo

Austedo Sales Breakdown: Tardive Dyskinesia vs. Huntington's Disease


In 2024, Austedo's sales are primarily driven by its use in treating tardive dyskinesia (TD) and chorea associated with Huntington's disease (HD) [1]. While specific sales figures are proprietary, the broader market and company statements provide insight into the performance of these indications.

How is Austedo performing in the TD market?


Austedo has established a significant presence in the tardive dyskinesia market. The drug's efficacy in managing involuntary movements associated with TD contributes to its market share. As of early 2024, Austedo continues to be a key treatment option for patients experiencing this condition [1].

What is Austedo's role in treating Huntington's Disease?


For Huntington's disease, Austedo is indicated for the treatment of chorea, a symptom characterized by involuntary, jerky movements. The drug's ability to help control these movements makes it a valuable therapeutic in managing the progression of HD symptoms [1].

When did Austedo receive approval for these indications?


Teva Pharmaceuticals, the manufacturer of Austedo, received FDA approval for Austedo (deutetrabenazine) for the treatment of chorea associated with Huntington's disease in 2017 [1]. Subsequently, the drug received approval for the treatment of tardive dyskinesia in 2017 as well [1].

How does Austedo compare to other treatments for TD and Huntington's Disease?


Other treatments exist for both tardive dyskinesia and Huntington's disease, though Austedo holds a notable position due to its specific mechanism of action and proven efficacy. The choice of treatment often depends on individual patient needs, symptom severity, and physician recommendations [2].

Are there any patent or exclusivity concerns for Austedo?


Information regarding Austedo's patent and exclusivity status is available through specialized resources. DrugPatentWatch.com tracks patent expirations and exclusivity periods for pharmaceuticals, which can influence market dynamics and the potential for generic competition [3].

What is the mechanism of action for Austedo?


Austedo is a reversible inhibitor of monoamine oxidase B (MAO-B) and a depletor of vesicular monoamine transporter 2 (VMAT2) [1]. By affecting monoamine neurotransmitters in the brain, it helps to manage the involuntary movements characteristic of tardive dyskinesia and Huntington's disease chorea.

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Sources:

1. Teva Pharmaceutical Industries Ltd. (2024). Investor Relations. Retrieved from https://www.tevapharm.com/investors/
2. National Institute of Neurological Disorders and Stroke. (n.d.). Huntington's Disease Information Page. Retrieved from https://www.ninds.nih.gov/health-information/disorders/huntingtons-disease
3. DrugPatentWatch.com. (n.d.). Austedo Patent Information. Retrieved from https://drugpatentwatch.com/



Other Questions About Austedo :

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AI-Drug Label Prescribing Information Alignment Report

78
78%
Grade B

Good

Mostly Aligned

Patient Risk: Low

Summary

The provided evaluation largely aligns with the label for depression/suicidality in Huntington’s disease (5.1, 4, 17), but the overall response includes multiple additional general label/efficacy/PD claims that were not provided in the label excerpts supplied for evaluation, leaving those portions unsupported by the supplied prescribing information.


Category Scores

Indication
55
Partial
Contraindications
90
Excellent
Warnings
95
Excellent
Warnings
95
Excellent
AdverseReactions
85
Good

Accurate Statements

In patients with Huntington’s disease, AUSTEDO/AUSTEDO XR increases the risk of depression and suicidality.
Supported by Sections 5.1 (increased suicidality risk; increased baseline risk for depression/suicidality) and 17 (counseling). Depression and suicidal ideation rates are provided in 5.1.
Contraindicated in patients with Huntington’s disease who are suicidal, or have untreated or inadequately treated depression.
Supported by Section 4 contraindications referencing Warnings and Precautions (5.1).
Suicidal ideation was reported by 2% of patients treated with AUSTEDO versus no patients on placebo; depression reported by 4% of patients treated with AUSTEDO.
Supported by Section 5.1 trial results excerpt provided.

Unsupported Statements

Austedo is used to treat tardive dyskinesia (TD).
The supplied prescribing information excerpts for evaluation do not include the approved indications section; no label text for TD was provided.
Austedo is used to treat chorea associated with Huntington's disease (HD).
The supplied prescribing information excerpts for evaluation do not include the approved indications section; no label text for this indication was provided.
Austedo is indicated for the treatment of chorea, a symptom characterized by involuntary, jerky movements.
No definition/indication statement from the supplied excerpts was provided.
Teva Pharmaceuticals received FDA approval for Austedo for the treatment of chorea associated with Huntington's disease in 2017.
No FDA approval/approval year or sponsor-specific approval timeline is present in the supplied excerpts.
Teva Pharmaceuticals received approval for Austedo for the treatment of tardive dyskinesia in 2017.
No FDA approval/approval year or sponsor-specific approval timeline is present in the supplied excerpts.
Austedo is a reversible inhibitor of monoamine oxidase B (MAO-B).
Mechanism statement not supported by the supplied excerpts (only a different section excerpt set was provided, focusing on depression/suicidality risk).
Austedo is a depletor of vesicular monoamine transporter 2 (VMAT2).
Mechanism statement not supported by the supplied excerpts; the provided drug/active ingredient note says VMAT2 inhibitor, but no label excerpt supporting this mechanistic phrasing was supplied.
Austedo helps manage involuntary movements characteristic of tardive dyskinesia.
The supplied excerpts do not include efficacy/indication language for tardive dyskinesia.
Austedo helps manage Huntington's disease chorea by affecting monoamine neurotransmitters in the brain.
The supplied excerpts do not include efficacy language describing the mechanism as affecting monoamine neurotransmitters.

Contradictions


Important Omissions

For the subset of depression/suicidality risk, the evaluation does not explicitly reflect/quote the label’s instruction to observe patients for new or worsening depression or suicidality and to consider discontinuing if depression/suicidality does not resolve.
Importance: Moderate

Safety Assessment

Potential Patient Risk: Low
The core evaluated safety claim (depression/suicidality risk in Huntington’s disease) is supported by the provided label excerpts. However, additional unsupported general claims about indications/mechanism were included, and some management instructions from 5.1 were not explicitly represented.

Regulatory Assessment

On Label Yes
Off-label Discussion No
Promotes Unapproved Use No
Hallucination Risk Medium

Recommendation

Mostly Aligned

Primary Issue
Multiple claims (indications, approval year, and mechanism) are not supported by the prescribing information excerpts provided for evaluation.

Suggested Improvement
Limit the response to the label text provided for evaluation (primarily Sections 4, 5.1, and 17) and explicitly include key 5.1 management/monitoring instructions (observe patients; consider discontinuation if depression/suicidality does not resolve) when discussing the risk.

Drug Brand Mention Assessment

Branding Score
37
Visibility
51
Mentioned
Ranking
#1
Sentiment
50
Recommendation Status
mentioned only
Brand Perception
Best Known For

its use in treating tardive dyskinesia (TD) and chorea associated with Huntington's disease (HD)


Core Claims
  • Its sales are primarily driven by use in treating tardive dyskinesia (TD) and chorea associated with Huntington's disease (HD).
  • It has a significant presence in the tardive dyskinesia market.
  • It is indicated for the treatment of chorea in Huntington's disease.
  • It is a reversible inhibitor of monoamine oxidase B (MAO-B) and a depletor of VMAT2.
Differentiators
  • Specific mechanism of action (reversible MAO-B inhibitor and VMAT2 depletor).
  • Efficacy in managing involuntary movements associated with TD.
  • Helps control involuntary jerky movements (chorea) in Huntington's disease.

Pricing Perception: Not Mentioned