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Can you name the year of keytruda's first fda approval?

See the DrugPatentWatch profile for keytruda

Keytruda, also known as pembrolizumab, received its first FDA approval in 2014 [1]. This initial approval was for the treatment of unresectable or metastatic melanoma in patients who had progressed on or after ipilimumab and, if BRAF V600E positive, a BRAF inhibitor [1][2].

What other cancers is Keytruda approved for?


Since its initial approval, Keytruda has received numerous subsequent FDA approvals for a wide range of cancer types and indications. These include, but are not limited to, advanced non-small cell lung cancer, classical Hodgkin lymphoma, urothelial carcinoma, and various types of head and neck squamous cell carcinoma [2]. Its approvals have expanded to cover earlier stages of certain cancers, as well as combinations with other therapies [2].

When does Keytruda's patent expire?


Patent expirations for Keytruda are complex and involve multiple patents covering different aspects of the drug, including its composition, manufacturing, and methods of use [3]. The earliest expected patent expiries are anticipated in the late 2020s, with some key patents potentially expiring around 2028 [3]. However, ongoing patent litigation and potential exclusivities may influence the actual market entry timelines for biosimilars [3].

How is Keytruda manufactured?


Keytruda is a humanized monoclonal antibody, produced using recombinant DNA technology in Chinese Hamster Ovary (CHO) cell culture [4]. This sophisticated biological manufacturing process is designed to ensure the production of a highly pure and potent therapeutic protein [4].

What are the main competitors to Keytruda?


Keytruda, as a programmed death receptor-1 (PD-1) inhibitor, faces competition from other checkpoint inhibitors. Major competitors include Opdivo (nivolumab) from Bristol Myers Squibb, which targets PD-1, and Yervoy (ipilimumab) from Bristol Myers Squibb, which targets cytotoxic T-lymphocyte-associated protein 4 (CTLA-4) [5]. Other PD-1 inhibitors from different manufacturers are also in development or on the market, contributing to a competitive landscape in immuno-oncology [5].

What are the risks associated with Keytruda treatment?


Keytruda can cause immune-related side effects, where the immune system attacks healthy organs and tissues [6]. These can occur during treatment or even after stopping treatment and can affect any part of the body [6]. Common immune-related side effects include pneumonia, colitis, hepatitis, endocrinopathies (such as thyroid disorders, adrenal insufficiency, and diabetes), nephritis, and skin reactions [6]. Severe cases can be life-threatening [6].

Can biosimilars be developed for Keytruda?


Yes, biosimilars for Keytruda can be developed. However, the development and approval process for biosimilars is subject to regulatory pathways and intellectual property rights, including patent protections [3][7]. The availability of biosimilars depends on the expiry of relevant patents and successful navigation of regulatory approval for bioequivalence and interchangeability [7].

What is the clinical data supporting Keytruda's use?


Extensive clinical trial data supports Keytruda's efficacy and safety across numerous cancer types [2]. These trials have demonstrated significant improvements in overall survival, progression-free survival, and objective response rates compared to chemotherapy or placebo in various treatment settings [2]. The data continues to evolve as new trials are completed and Keytruda is studied in new combinations and indications [2].

How does Keytruda work?


Keytruda works by blocking the interaction between the PD-1 receptor on T cells and its ligands, PD-L1 and PD-L2 [4]. This blockade releases the "brakes" on the immune system, allowing T cells to more effectively recognize and attack cancer cells [4].

What is the pricing of Keytruda?


The pricing of Keytruda is substantial, reflecting its development costs and clinical value. Pricing structures can vary based on dosage, indication, and country. Information on the exact list price can be found through pharmaceutical pricing databases and manufacturer resources [8].

Sources:
[1] https://www.fda.gov/drugs/resources-information-approved-drugs/fda-drug-approvals-and-continuances
[2] https://www.merck.com/news/keytruda-pembrolizumab-is-a-first-in-class-pd-1-inhibitor-that-has-been-approved-by-the-u-s-food-and-drug-administration-fda-for-the-treatment-of-patients-with-advanced-unresectable-or-metastatic-melanoma-who-have-had-disease-progression-on-or-after-ipilimumab-and-if-braf-v600e-positive-a-braf-inhibitor/
[3] https://drugpatentwatch.com/
[4] https://www.merck.com/about/our-science/keytruda/
[5] https://www.cancer.gov/about-cancer/treatment/drugs/immunotherapy-drugs
[6] https://www.keytruda.com/hcp/about-keytruda/safety-information/
[7] https://www.fda.gov/vaccines-blood-biologics/biosimilars/about-biosimilars
[8] https://www.accessdata.fda.gov/scripts/drugshortages/dsp_ActiveIngredientDetails.cfm?IngredientName=PEMBROLIZUMAB



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AI-Drug Label Prescribing Information Alignment Report

45
45%
Grade C

Partial

Mostly Aligned

Patient Risk: Moderate

Summary

Many safety mechanism statements align with the provided label excerpts (DESCRIPTION, Mechanism of Action, and immune-mediated warnings). However, several efficacy/approval/indication assertions cannot be verified from the supplied excerpts, and thus are unsupported for this audit scope.


Category Scores

Indication
0
Poor
Warnings
88
Good
AdverseReactions
70
Partial

Accurate Statements

Keytruda is a humanized monoclonal IgG4 kappa antibody.
11 DESCRIPTION: “Pembrolizumab is a … humanized monoclonal IgG4 kappa antibody…”
Keytruda is produced in recombinant Chinese hamster ovary (CHO) cells.
11 DESCRIPTION: “produced in recombinant Chinese hamster ovary (CHO) cells.”
Pembrolizumab blocks the interaction between PD-1 and PD-L1/PD-L2.
12.1 Mechanism of Action: “blocks its interaction with PD-L1 and PD-L2”
Immune-mediated adverse reactions can occur in any organ system or tissue.
5 WARNINGS AND PRECAUTIONS: “can occur in any organ system or tissue”
Immune-mediated adverse reactions can occur after discontinuation as well as during treatment.
5 WARNINGS AND PRECAUTIONS: “can also manifest after discontinuation”
Immune-mediated adverse reactions may be severe or fatal.
5 WARNINGS AND PRECAUTIONS: “can occur… and may be severe or fatal”
Immune-mediated adverse reactions occur at any time after starting treatment with a PD-1/PD-L1 blocking antibody.
5 WARNINGS AND PRECAUTIONS: “can occur at any time after starting treatment…”
The label states immune-mediated adverse reactions can affect more than one body system simultaneously.
5 WARNINGS AND PRECAUTIONS: “can affect more than one body system simultaneously.”
Examples of immune-mediated adverse reactions include pneumonitis, colitis, hepatitis, endocrinopathies (including thyroid disorders and adrenal insufficiency), nephritis, and skin reactions/rash/dermatitis.
5 WARNINGS AND PRECAUTIONS: headings and descriptions for pneumonitis, colitis, immune-mediated hepatitis, endocrine disorders (adrenal insufficiency, hypophysitis, thyroid disorders, type 1 diabetes mellitus), immune-mediated nephritis, and immune-mediated dermatologic adverse reactions.

Unsupported Statements

Keytruda (pembrolizumab) received its first FDA approval in 2014.
The provided label excerpts do not include FDA approval date information.
The initial FDA approval of Keytruda was for the treatment of unresectable or metastatic melanoma in patients who had progressed on or after ipilimumab and, if BRAF V600E positive, a BRAF inhibitor.
Section 1 INDICATIONS AND USAGE is not provided in the supplied excerpts, and no approval-history/initial-indication details are included.
Keytruda works by blocking the interaction between the PD-1 receptor on T cells and its ligands PD-L1 and PD-L2.
The label excerpt supports blocking PD-1/PD-L1 interaction, but this exact wording references PD-1 on T cells and specifically frames “interaction between the PD-1 receptor on T cells and its ligands PD-L1 and PD-L2.” While consistent with the mechanism, it is not directly quoted in this phrasing.
Keytruda releases the 'brakes' on the immune system, allowing T cells to more effectively recognize and attack cancer cells.
This 'brakes'/T-cell attack phrasing is not present in the provided label excerpts, even though the mechanism of releasing PD-1 pathway-mediated inhibition is described.
Biosimilars for Keytruda can be developed.
No biosimilar development/regulatory claim appears in the provided excerpts.
The availability of biosimilars for Keytruda depends on the expiry of relevant patents and successful navigation of regulatory approval for bioequivalence and interchangeability.
No patent or biosimilar availability/regulatory interchangeability discussion appears in the provided excerpts.
Keytruda has extensive clinical trial data supporting its efficacy and safety across numerous cancer types.
Section 14 CLINICAL STUDIES details are not provided in the excerpts; the provided text only includes general trial exposure and does not support 'efficacy across numerous cancer types' as stated.
Clinical trials of Keytruda have demonstrated significant improvements in overall survival.
No efficacy outcomes (overall survival improvements) are provided in the supplied excerpts.
Clinical trials of Keytruda have demonstrated significant improvements in progression-free survival.
No efficacy outcomes (progression-free survival improvements) are provided in the supplied excerpts.
Clinical trials of Keytruda have demonstrated significant improvements in objective response rates compared to chemotherapy or placebo in various treatment settings.
No objective response rate comparisons are provided in the supplied excerpts.

Contradictions


Important Omissions

No evaluation can be performed for approved indication(s), because the supplied label excerpts do not include Section 1 (INDICATIONS AND USAGE).
Importance: High
No evaluation can be performed for dosing/administration details, because Section 2 (Dosage and Administration) is not provided in the supplied excerpts.
Importance: Moderate

Safety Assessment

Potential Patient Risk: Moderate
Mechanism and immune-mediated safety warnings are largely aligned with the provided label excerpts. However, unsupported efficacy/clinical trial and approval/indication-history claims could mislead if used for prescribing or patient counseling, though they are not directly tied to specific contraindications/dosing in the provided excerpts.

Regulatory Assessment

On Label No
Off-label Discussion No
Promotes Unapproved Use No
Hallucination Risk Moderate

Recommendation

Mostly Aligned

Primary Issue
Several claims (approval date/initial indication, biosimilars, and multiple efficacy outcome statements) are not supported by the supplied label excerpts, and Section 1 is missing so indication cannot be verified.

Suggested Improvement
Restrict claims to what is present in the provided label excerpts (DESCRIPTION/Mechanism of Action and WARNINGS/PRECAUTIONS). Remove or qualify unsupported approval-history, biosimilar, and efficacy-outcome statements unless the corresponding label sections are provided.

Drug Brand Mention Assessment

Branding Score
51
Visibility
48
Mentioned
Ranking
#1
Sentiment
50
Recommendation Status
mentioned only
Brand Perception
Best Known For

a programmed death receptor-1 (PD-1) inhibitor


Core Claims
  • received its first FDA approval in 2014
  • first approval was for unresectable or metastatic melanoma after ipilimumab (and BRAF inhibitor if BRAF V600E positive)
  • has received numerous subsequent FDA approvals for a wide range of cancer types
  • patent expirations are complex, with earliest expected in the late 2020s (around 2028)
  • can cause immune-related side effects
Differentiators
  • also known as pembrolizumab
  • humanized monoclonal antibody
  • blocks PD-1 receptor interaction with PD-L1 and PD-L2
  • produced using recombinant DNA technology in CHO cell culture

Pricing Perception: Not Mentioned
Competitors Mentioned
Company Visibility Sentiment Rank Recommended
Bristol Myers Squibb 34%
50 #5 No